• Diagnostic and Therapeutic Aspects of Hemiplegic Migraine

• Brain atrophy following hemiplegic migraine attacks

• The de novo CACNA1A pathogenic variant Y1384C associated with hemiplegic migraine, early-onset cerebellar atrophy and developmental delay leads to a loss of Cav2.1 channel function

• The clinical spectrum of familial hemiplegic migraine associated with mutations in a neuronal calcium channel

• CACNA1A mutation linking hemiplegic migraine and alternating hemiplegia of childhood

• Recurrent Stroke Due to a Novel Voltage Sensor Mutation in Cav2.1 Responds to Verapamil | Stroke

• Zebrafish as a Model System for the Study of Severe CaV2.1 (α1A) Channelopathies

The resources on this page are evolving as our panel makes progress on a consensus for treatment guidelines. Given the potential consequences of CACNA1A-related hemiplegic migraines, we felt a sense of urgency to provide an immediate resource for families and their physicians. Please return to this page for updated guidelines and additional resources in the near future. The contents of this guide are not intended to substitute for professional medical advice, diagnosis or treatment. Please consult your physician for personalized medical advice.